第一届年度 STEM 写作比赛的获奖名单

从墨西哥辣椒和口香糖到癌症治疗和黑洞,学生们解释了科学、技术、工程和数学领域的概念。

。。。史蒂夫·格施迈斯纳/科学来源

你有没有想过为什么世界不会每次眨眼都变黑?或者说,无能的人怎么会对自己的能力如此自信?或者海洋中的塑料对我们的食物意味着什么?

这些学生有答案。

在我们有史以来的首届STEM写作比赛中,学习网络与《科学新闻》合作,挑战青少年选择一个他们感兴趣的与STEM相关的问题,概念或问题,并以500字或更少的字数向普通观众解释它清晰引人入胜的方式。

我们收到了1,618份参赛作品,涉及医学和心理学,化学和生物学,几何学和天文学等主题。其中,我们的评委,包括《纽约时报》的科学记者和全国各地的科学教育工作者,选出了44名决赛选手——8名获奖者、14名亚军和22名荣誉奖——我们在下面列出了他们。

我们从来都不确定像这样的新比赛会发生什么,但我们希望学生们能以探究和发现的精神来承担这项任务。他们做到了。

我们的一位获奖者告诉我们,在注意到蚁丘主宰着她的后院后,她受到启发,开始研究蚂蚁状微型机器人。另一个人想知道是什么导致了她的许多朋友和家人遭受的睡眠呼吸暂停。一位亚军质疑是什么让辛辣食物如此吸引人,辛辣食物是她墨西哥传统的主食。

为了找到答案,他们搜索了新闻文章和科学期刊,采访了专家,甚至进行了自己的实验。

但是,让我们的获奖者与众不同的不仅仅是他们写了什么或他们用来支持它的证据——而是他们巧妙地解释他们的主题,以便普通观众能够理解它们的方式。毕竟,这是一场写作比赛。凭借引人入胜的钩子、相关的类比、巧妙的隐喻和强烈的声音感,这些作家不仅帮助翻译了复杂的主题;他们还使他们阅读愉快。

但不要相信我们的话。我们将全文发布八篇获奖论文,您可以通过单击下面的作品链接自行阅读。我们希望,像我们一样,你会学到新的东西,并从中获得乐趣。

感谢所有参与的学生,并祝贺我们所有的决赛选手。如果您对本次比赛有任何反馈,请写信给我们 LNFeedback@nytimes.com。

按作者姓氏的字母顺序排列。

获奖评论

A New Cancer Treatment That Is Faster, More Efficient and Less Risky Is Coming: Bacteria Bombs” by William Chesney

Fat Got Your Tongue?” by Nicole Fang

Circadian Rhythms: The Conductor of Our Body’s Symphony” by Aliya Fisher

The Dunning-Kruger Effect: Why Incompetence Begets Confidence” by Allison He

An Unexpected Dinner Guest: Marine Plastic Pollution Hides a Neurological Toxin in Our Food” by Vivian Li

Quorum Sensing: What We Can Learn From Eavesdropping on Bacteria” by Jenny Mao

In the Blink of an Eye” by Rivka Shields

Tiny in Size, but Goliath in Force: The Colonization of Ant-Based Microrobotics” by Celina Zhao

“Spice It Up” by Sofia Gutierrez Boker

“Telomeres — Turning Back the Biological Clock” by Hubert Chen

“The Promise of Hot Garbage” by Michael Dekhtyar

“The Death of Classical Geometry” by Elias Leventhal

“Egg Stem Cells May Mean the Creation of New Eggs After Birth” by Erin Li

“How the Phishermen of Today Cast Their Lines” by Amy Liu

“Far-off Galaxy Collision Foretells the ‘Tail’ of Our Future” by Diya Naik

“Gravitational Waves and General Relativity” by Maximilian Niebur

“A New Hope for Bees: Genetically Modified Gut Bacteria” by Eva Panin

“A Zombie Apocalypse?” by Tisya Raina

“Is it the End for the Silver Bullet?” by Jia Cheng Anthony Shen

“Sizzling Steaks and Crusty Bread: The Science of the Maillard Reaction” by Victoria Tong

“Black Holes: The Universe’s Biggest Subwoofers” by Will

“Can We Really Heal Ourselves? Demystifying Placebos” by Phoebe Yu

“Could Hydrogen Fuel Cell Vehicles Be the Future of Transportation?” by Quinn Alami

“What Are the Effects of Delaying School Start Times on Teens?” by Aylin Ardali

“Should We Trust Our Memories?” by Jessie Gaither

“No, Amazon Does Not Produce 20% of the World’s Oxygen. Our Breath Is a Gift From the Wanderers-on-the-Oceans” by Devanshi Guglani

“Using Rat Urine and Chewing Gum Science Could Thwart Climate Change” by Kwangjun Jung

“Must-Fix-It: Energy Poverty” by Logan Kim

“The Science Behind Tattoos” by Julia Luyk

“The Future of Cancer Imaging Lies in The Starry Night Under the Sea” by Camilla Martinez

“How Microbes Decide Whether We Live or Die” by Gus Morrill

“The Unexpected Legacy of Our Digital Founding Fathers” by Ellee Nakamura

“Forensics Fiction? The Reliability of Facts in Our Courtrooms” by Mackenzie Pavlik

“How Caffeine Can Help You Stick to Your New Year’s Resolutions for Longer” by Joshua Pixley

“Premature Glaucoma” by Marina Lupercio Sanchez

“The Rusty Patched Bumblebee: A Ticking Time Bomb” by Erin Saunders

“Marshmallow Experiment — True or All Fluff?” by Rose Sawilowsky

“The Magic Behind Flu Vaccines — Secret Weapons Against Influenza Virus in the Everlasting Arms Race” by Yihan Shen

“Making Toilets Sustainable” by Ana-Maria Skaricic

“Keeping You in the Loop About Your Poop” by Kenna Sondhelm

“Learning? Sleep On It.” by Heather Szczesniak

“The Effects of Noise Pollution on Marine Wildlife” by Hadley Weathers

“Why Gray Matter, Matters: Inside the Brain of a Psychopath” by Paige Williams

“Being Sustainable After Death” by Tiffany Wu


From the New York Times Science and Well sections: David W. Brown, Cara Giaimo, Dennis Overbye, Tara Parker-Pope, Marion Renault, Nicholas St. Fleur and Shannon Stirone

From The Learning Network: Jeremy Engle, Michael Gonchar and Natalie Proulx

Other judges, including science educators from schools and education organizations around the country: Victoria Bampoh, Amanda Christy Brown, Terianne Hall, Annissa Hambouz, Meghan Hess, Shira Katz, Allison Lee, Simon Levien, Keith Meatto, Mariam Naraine, Dawn Parker, Melissa Slater and Vanessa Vieux

Tiny in Size, but Goliath in Force: The Colonization of Ant-Based Microrobotics

我们通过发表论文来表彰学生 STEM 写作比赛的前八名获奖者。这是赵淑娴的。

这篇文章由来自佐治亚州雅典北奥科尼高中的17岁的Celina Zhao撰写,是学习网络有史以来第一届STEM写作比赛的前八名获奖者之一,我们收到了1,618份参赛作品。

Tiny in Size, but Goliath in Force: The Colonization of Ant-Based Microrobotics

Imagine climbing a skyscraper — but while lugging an elephant. Or with a team of five others, lifting the weight of the Eiffel Tower and three Statues of Liberty. As Sisyphean as this sounds, functional equivalents of these incredible feats have already been accomplished by ant-inspired microrobots. And these examples are only the tip of the anthill.

As the ants keep marching on, dominating as a massive superorganism that has colonized nearly every landmass on Earth, researchers are increasingly turning toward ants as inspiration for biomimicry-based robots. An army of ants boasts the ideal physical anatomy and biological organization for transforming the capabilities of microrobots in the real world — from leading in search-and-rescue missions, to mapping previously unexplored terrain on earth and in space.

At the base level, simple math and physics govern the tremendous strength of the individual ant. From the square-cube law, ants have an optimum ratio between surface area and volume: analogous to the ratio between strength and mass. This allows ants to exert more force per milligram of weight. The physiology of ants are another asset to explore. Researchers at Ohio State University reported on the astonishing power of an ant’s neck joint in the Journal of Biomechanics in 2014. By measuring how much centrifugal force was required to separate the ant’s body from its head (the same force you feel on “rotor rides” at the carnival — minus the decapitation, of course), they discovered that the joint could support 3,400 to 5,000 times the body weight of the ant!

But their biological and social coordination is what really sets ants apart. Not only are ants able to pool their strength into a collective unit, but they also practice decentralized communication through chemical signals called pheromones. This protean system functions similarly to a chain of ripples in water, and it effectively allows for cohesive teamwork even across wide distances.

When a combination of these abilities is applied to swarm robotics, the results are impressive. MicroTug robots created at Stanford University are one example. They made headlines when a team of six — weighing just 3.5 ounces together — pulled a car weighing 3,900 pounds. Synchronizing the bots’ movements so that each used three of their six legs, much like how ants cooperate in real life, made this act possible. Tribots, an invention of Switzerland’s École Polytechnique Fédérale de Lausanne (EPFL) and Osaka University, are another instance. Based on trap-jaw ants, division of labor and leadership is a core component of Tribots. So, if one robot fails, the system of “fault tolerance” means that the success of the mission won’t be adversely affected. Both MicroTug and Tribot robots hold immense potential in emergency relief and exploration and navigation missions.

“Just like insects, small robots can be powerful,” says Jamie Paik, lead researcher at EPFL, echoing a growing sentiment among the research world. And given the breadth of ant abilities still untapped by microrobots, it won’t be long before there are more queen robots in the field.

Works Cited

Akpan, Nsikan. “How Do Ants Synchronize to Move Really Big Stuff?” PBS NewsHour, 28 July 2015.

“Ant Factoids.” Arizona State University.

Gorder, Pam Frost. “Study of Ants’ Remarkable Strength May Lead to Powerful Micro-Sized Robots.” The Ohio State University, 13 Feb. 2014.

Gordon, Deborah M. “Local Links Run the World.” Aeon, 1 Feb. 2018.

Hernandez, Daisy. “Robotic Ants Function Just Like Real Ones.” Popular Mechanics, 15 July 2019.

Johnson, George. “Of Mice and Elephants: A Matter of Scale.” 12 Jan. 1999.

Koenig, Sven. “Tutorial on Ant Robotics.” Sven Koenig.

Markoff, John. “Modeled After Ants, Teams of Tiny Robots Can Move 2-Ton Car.” The New York Times, 13 March 2016.

“Microtugs.” Stanford Biomimetics and Dexterous Manipulation Lab, 28 April 2016.

Stevens, Allison Pearce. “Tiny Microrobots Team up and Move Full-Size Car.” ScienceNews for Students, 19 April 2016.

In the Blink of an Eye

我们通过发表论文来表彰学生 STEM 写作比赛的前八名获奖者。这是Rivka Shields的。

Fulvio De Filippi/Getty Images

这篇文章由Rivka Shields撰写,来自大华盛顿州的Y eshiva - 马里兰州银泉的Tiferes Gedaliah,是学习网络有史以来第一次STEM写作比赛的前八名获奖者之一,我们收到了1,618份参赛作品。

In the Blink of an Eye

Imagine walking into a theater to watch a movie. While it is playing, the screen goes blank every ten seconds. Wouldn’t it ruin the movie? Humans blink about six times per minute, a blink every 10 seconds. Why do we not notice the world going black every time our eyelids close?

Blinking, also known as the cornea reflex, is our brains’ automatic response when anything comes too near our eyes that could potentially be damaging. Our eyelids have tear film, made from fluids and oils, on them. Blinking spreads those over the outer eye, cleaning and moisturizing it. This is essential, or the eyes will dry out. In fact, it is so important that for patients with nerve damage that prevents them from closing their eyelids, doctors are sewing small gold weights into their eyelids so they will close easier to lubricate the eye. The question still remains, though. Why don’t we notice that the world is going black for about 400 milliseconds, the average blinking time?

Before we blink, we signal to our brain that we are about to blink. Our brains take a “picture” of what we are seeing now, and save it in our brain. We hold that picture for the milliseconds that our eyes are closed, so when we open our eyes again, it seems that nothing has changed. This is why our vision is so seamless; we still have a picture in the mind of what we are looking at. When we blink, our eyeballs roll backward and may not get back to their original positions when our eyes open again. The brain compares the “pictures” of before the blink and after the blink, and forces our eye muscles to fill in the gaps.

A study was done by the Nanyang Technology University in Singapore in a dark room with a dot on a screen. Cameras tracked their blinks, so that every time they blinked, the dot moved one centimeter. Participants didn’t notice the movement, as their brains immediately readjusted to the dot. After about 30 of these dot movements, researchers noticed that the participants’ eyes immediately shifted as they blinked, to anticipate where their brain expected the dot to be. This caused the participants to not even be aware that they blinked, because when their eyes reopened, their eye muscles immediately knew what had changed and filled in for the lost time.

Understanding how the brain works together with the eyes to make up for changes outside the body during a blink is important, because it could help scientists find ways for the brain to make up for other changes inside or outside the brain. For example, if an eye were injured, they now know how the brain can make up for lost vision. Gerrit Maus, an assistant professor of psychology at Nanyang Technological University in Singapore, said, “These findings add to our understanding of how the brain constantly adapts to changes, commanding our muscles to correct for errors in our bodies’ own hardware.”

Works Cited

Laliberte, Marissa. “Ever Wondered Why Things Don’t Get Darker When We Blink?” The Healthy, 9 Feb. 2017.

“Science Watch: Repairing the Blink.” The New York Times, 7 Aug. 1990.

Stern, John. “Why Do We Blink?” NBC News, 25 Feb. 1999.

Stromberg, Joseph. “Why Do We Blink So Frequently?” Smithsonian Magazine, 24 Dec. 2012.

“Why the Lights Don’t Dim When We Blink.” ScienceDaily, 19 Jan. 2017.

Quorum Sensing: What We Can Learn From Eavesdropping on Bacteria

我们通过发表论文来表彰学生 STEM 写作比赛的前八名获奖者。这是珍妮·毛(Jenny Mao)的作品。

这篇文章由来自加利福尼亚州圣马特奥卡尔蒙特高中的 17 岁的 Jenny Mao 撰写,是学习网络首届 STEM 写作比赛的前八名获奖者之一,我们收到了 1,618 份参赛作品。

Quorum Sensing: What We Can Learn From Eavesdropping on Bacteria

The sun has long disappeared over the horizon, but a deep glow emits from inside the ocean. The shimmering water takes on an unnerving shade of electric blue. To many mariners over the past centuries, this rare occurrence was proof that the ocean had fantastic creatures hidden in its depths that we had yet to explore and understand. And they weren’t entirely incorrect.

This phenomenon, known as the milky seas, is due to the bioluminescence of the bacteria Vibrio fischeri. In 1970, the scientists Kenneth H. Nealson and John W. Hastings of Harvard University were running tests on these vibrant Vibrio fischeri bacteria when they noticed something rather strange. A lone bacteria, in dilute suspension, would not emit any light. However, when allowed to grow to a certain cell population size, they would all light up, as if someone had flipped the switch on a string of Christmas lights.

It’s as if the simple bacteria are able to coordinate and act in perfect synchronicity with each other! How can this be? Welcome to the world of quorum sensing. Essentially, quorum sensing is the system that bacteria use to communicate with one another and regulate their gene expression based on population density.

To understand why bacteria use quorum sensing, imagine this: A single bacteria invading a host body doesn’t stand a good chance of succeeding because the immune system can easily stop a low-volume attack. So, it’s in that bacteria’s best interest to wait quietly until it knows that there are enough bacteria accompanying it. Then, they can virulently infect the host cell all at once. Sneaky, but effective. How do they do it?

Essentially, like humans, bacteria have their own languages. They communicate using chemical signals known as autoinducers. Autoinducers are secreted by the bacteria, and once the autoinducers are present at a high enough concentration, they can activate the transcription of certain genes, such as luminescence. In fact, according to research done by Stephan Schauder and Bonnie L. Bassler, each bacteria type uses a slightly different autoinducer so that the messages don’t get mixed up. However, they also found that there is a common five-carbon molecule that is produced by every single bacterium. Dr. Bassler dubs it the “bacterial Esperanto” because it is used to communicate among different species of bacterium!

The bacterial Esperanto and common bacterial sensors may be the future of combating viruses, especially as bacteria begin to develop antibiotic immunity. As Andrew Pollack writes in his New York Times article, “Drug Makers Listen in While Bacteria Talk,” researchers are working to design molecules that will clog up bacterial sensors. Like putting on noise-canceling headphones, these molecules would muffle the impact of the signaling molecules, limiting the ability for virulent bacteria to communicate.

Quorum sensing is a fascinating framework that can help explain glowing oceans and nasty colds, as well as having promising implications in fighting infections and antibiotic immunity. The bacteria are talking. All we have to do is listen.

Works Cited

González, Juan E., and Neela D. Keshavan. “Messing With Bacterial Quorum Sensing.” American Society for Microbiology: Microbiology and Molecular Biology Reviews, Dec. 2006.

Miller, Steven D., Steven H. D. Haddock, Christopher D. Elvidge and Thomas F. Lee. “Detection of a Bioluminescent Milky Sea From Space.” Proceedings of the National Academy of Sciences of the United States of America, 4 Oct. 2005.

Pollack, Andrew. “Custom-Made Microbes, at Your Service.” The New York Times, 17 Jan. 2006.

Pollack, Andrew. “Drug Makers Listen In While Bacteria Talk.” The New York Times, 27 Feb. 2001.

Rutherford, Steven T., and Bonnie L. Bassler. “Bacterial Quorum Sensing: Its Role in Virulence and Possibilities for Its Control.” Cold Spring Harbor Perspectives in Medicine, Nov. 2012.

Schauder, Stephan, and Bonnie L. Bassler. “The Languages of Bacteria.” Genes and Development, 2001.

An Unexpected Dinner Guest: Marine Plastic Pollution Hides a Neurological Toxin in Our Food

我们通过发表论文来表彰学生 STEM 写作比赛的前八名获奖者。这是李薇薇的。

。。。李薇薇
这篇文章由来自马里兰州北波托马克市蒙哥马利布莱尔高中的 16 岁的 Vivian Li 撰写,是学习网络首届 STEM 写作比赛的前八名获奖者之一,我们收到了 1,618 份参赛作品。

An Unexpected Dinner Guest: Marine Plastic Pollution Hides a Neurological Toxin in Our Food

In the mid-1950s, domesticated cats in Minamata, Japan, mysteriously began to convulse and fall into the bay. The people of Minamata took on similar symptoms shortly after, losing their ability to speak, move and think.

Chisso Corporation, a Japanese chemical company, had dumped more than 600 tons of mercury into the bay between 1932 and 1968 via their wastewater. Over the next several decades, 1,784 people living near the shore died of shared uncanny symptoms while doctors scrambled to uncover the cause.

The Minamata Bay disease characterizes long-term impairment of the central nervous system from methylmercury poisoning. Although government organizations worldwide have since limited the mercury that enters surface waters, this toxin has a new and powerful avenue to the human brain: marine plastic pollution.

Methylmercury journeys up the food chain from phytoplankton and zooplankton to fish and humans. Dr. Katlin Bowman, a research scientist at the University of California, Santa Cruz, explains that heavy metal toxins naturally adhere to plastics in the water, creating extremely concentrated “fish food bombs” of mercury. “Plastic has a negative charge and mercury has a positive charge. Opposites attract so the mercury sticks,” Dr. Bowman said.

According to Abigail Barrows, a marine research scientist from the College of the Atlantic, microplastics are even more concentrated in methylmercury as a result of their greater ratio of surface area to volume, trapping toxic particles in the many folds and tight spaces. Less than five millimeters in size, microplastics range from microbeads in personal care products to microfibers from clothing. “If microplastics increase the rate of methylmercury production, then microplastics in the environment could indirectly be increasing the amount of mercury that accumulates in fish,” Dr. Bowman said.

Two key characteristics worsen methylmercury’s impact: bioaccumulation and biomagnification. With bioaccumulation, “the longer the fish lives, it keeps eating mercury and doesn’t lose it, so it ends up concentrating very high levels of mercury in its tissues,” said Dr. Nicholas Fisher, a distinguished professor at the State University of New York Stony Brook. “The methylmercury also biomagnifies, meaning the concentration is higher in the predator than it is in the prey.” Predators at the top of the food chain have more than 100,000 times more methylmercury in their bodies compared to the surrounding water.

However, our focus should be on the plastic pollution issue rather than mercury discharge. Dr. Carl Lamborg, a research scientist at U.C. Santa Cruz, explains that while mercury naturally cycles through the environment, plastics serve as a magnet, prolonging its lifetime in the ocean and funneling it into the mouths of plankton and fish.

The Minamata Bay disaster has already spelled out the horrific effects of mercury poisoning in all of its nitty-gritty glory, but the 87,000 tons of plastic in the growing Great Pacific Garbage Patch ensures that the problem will only swell.

“The plastic produced is on trend to double in the next 20 years,” said Dr. Barrows. “So I think that’s where we need to focus on in terms of worrying about our environment.”

Works Cited

Albeck-Ripka, Livia. “The ‘Great Pacific Garbage Patch’ Is Ballooning, 87,000 Tons of Plastic and Counting.” The New York Times, 22 Mar. 2018.

“A Short Explanation of the Mercury Issue.” European Commission, 7 Dec. 2012.

Barboza et al. “Microplastics Cause Neurotoxicity, Oxidative Damage and Energy-Related Changes and Interact with the Bioaccumulation of Mercury in the European Seabass, Dicentrarchus Labrax (Linnaeus, 1758).” Aquatic Toxicology, Feb. 2018.

Barrows, Abigail. Marine Research Scientist, College of the Atlantic. Interview.

Bowman, Katlin. Postdoctoral Research Scholar, University of California Santa Cruz. Interview.

Fisher, Nicholas. Distinguished Professor, State University of New York Stony Brook. Interview.

Lamborg, Carl. Associate Professor, University of California Santa Cruz. Interview.

Liboiron, Max. “Plastics & Methylmercury.” Civic Laboratory for Environmental Action Research, 15 July 2017.

“Minamata Disease.” Boston University Sustainability.

NOAA. “What Are Microplastics?” National Ocean Service website, 13 Apr. 2016.

Winner, Cherie. “How Does Toxic Mercury Get into Fish?” Oceanus Magazine, 1 Oct. 2010.

The Dunning-Kruger Effect: Why Incompetence Begets Confidence

我们通过发表论文来表彰学生 STEM 写作比赛的前八名获奖者。这是何炳强的。

。。。克里斯托弗·弗隆/盖蒂图片社
这篇文章由来自德克萨斯州丹顿市德克萨斯数学与科学学院的 17 岁的 Allison He 撰写,是学习网络首届 STEM 写作比赛的前八名获奖者之一,我们收到了 1,618 份参赛作品。

The Dunning-Kruger Effect: Why Incompetence Begets Confidence

Meet Dave. After a year of creating campaigns for a marketing company, Dave is convinced that his advertising skills are the best in his department. In his mind, his incredibly original content warrants a nice bonus. But when his manager hands him an annual review, his face falls. “This isn’t right,” Dave says, incredulous. He points at the section that marks him at the bottom 25 percent of employee performance and asks if it’s a mistake.

Dave’s case illustrates a psychological phenomenon called the Dunning-Kruger effect.

The Dunning-Kruger effect, coined by the psychologists David Dunning and Justin Kruger in 1999, is a cognitive bias in which poor performers greatly overestimate their abilities. Dunning and Kruger’s research shows that underperforming individuals “reach erroneous conclusions and make unfortunate choices, but their incompetence robs them of the ability to realize it.” This incompetence, in turn, leads them to “hold inflated views of their performance and ability.”

To reach these findings, Dunning and Kruger conducted a study that tested participants’ abilities in humor, logical reasoning and grammar. Before showing the participants their scores, Dunning and Kruger had them judge their performance on a percentile scale. What they found across all three categories confirmed their prediction of self-inflated assessment among underperforming individuals: participants scoring around the 15th percentile evaluated their percentile placements to be approximately 50 percent higher. And although results also indicated a slight overestimation by the average-scorers and a slight underestimation by the top-scorers, Dunning and Kruger focused on investigating the substantial overestimation by the bottom-scorers.

Were the underperforming individuals unable to recognize competence due to their own lack of it? To address this, Dunning and Kruger invited back the bottom-scoring and top-scoring participants and gave them the other participants’ tests to grade. Afterward, the participants were asked to re-evaluate their original testing score. The bottom-scorers, instead of recognizing their underperformance and lowering their rank accordingly, continued to elevate their scores. This suggested that the bottom-scorers needed competence to perceive competence.

If it takes competence to be aware, how are underperformers supposed to recognize their own lack of ability? The answer is in the question itself. Because it takes competence to be aware, underperformers must become competent. After training the bottom-scorers in logical reasoning, Dunning and Kruger discovered not only that their scores improved, but also that they no longer inflated their scores. These results indicate that knowledge and experience are crucial for gaining both ability and self-awareness.

In essence, we can’t reach a high level of competence without actively seeking feedback and knowledge. And for any activity — whether it’s marketing, parenting, football or underwater basket-weaving — it takes an open mind to gain the experiences that help us see our mistakes and grow from them.

The hallmark of intelligence, according to Dunning, is being “good at knowing what we don’t know.” If we want to avoid the impact of cognitive biases such as the Dunning-Kruger effect and if we want to better ourselves overall, it’s vital that we’re aware that there is always more to know.

Works Cited

Cherry, Kendra. “The Dunning-Kruger Effect.” Verywell Mind, 14 June 2019.

Kruger, Justin, and David Dunning. “Unskilled and Unaware of it: How Difficulties in Recognizing One’s Own Incompetence Lead to Inflated Self-Assessments.” Journal of Personality and Social Psychology, Dec. 1999.

Morris, Errol. “The Anosognosic’s Dilemma: Something’s Wrong but You’ll Never Know What It Is (Part 1).” The New York Times, 21 June 2010.

Circadian Rhythms: The Conductor of Our Body’s Symphony

我们通过发表论文来表彰学生 STEM 写作比赛的前八名获奖者。这是Aliya Fisher的作品。

。。。伊奇诺里

这篇文章由来自纽约布朗克斯布朗克斯布朗克斯科学高中的16岁的Aliya Fisher撰写,是学习网络有史以来第一届STEM写作比赛的前八名获奖者之一,我们收到了1,618份参赛作品。

Circadian Rhythms: The Conductor of Our Body’s Symphony

Our bodies are like musical compositions, different instruments completing the whole piece. The heart is the percussion, keeping pace for the rest of our bodies. The digestive system is the bass, providing the foundation everything else body systems need. The brain is the piano, giving us life and coordination. But who is the conductor? To whom do we owe the perfect synchronization of all instruments? The mastermind may come as a surprise, but the miracle-worker of our bodies’ symphony is the circadian clock — a series of molecular cues which integrate external stimuli to keep our bodies’ cycles on a 24-hour schedule.

Our mastermind’s lair is a compartment of the brain called the suprachiasmatic nucleus (S.C.N.), containing only 50,000 of the 86 billion neurons in our body. Our eyes detect light, signaling directly to the S.C.N., as if the conductor were being cued by the mood of the audience. During the day, high intensity light signals the S.C.N. to produce the proteins period (Per) and cryptochrome (Cry), which in turn activate myriad downstream pathways controlling metabolism, sleep and overall activity level. Meanwhile, Per and Cry accumulate in the cytoplasm, reaching critical levels at which they translocate to the nucleus and bind to receptors that shut down the production of Per and Cry. This slows metabolic activity and prepares the body for sleep.

This diurnal feedback loop is the crux of homeostasis, or biological equilibrium, but all musicians know what can happen when the conductor of the orchestra misses a beat. For instance, when we experience jet lag after flying between different time zones, our circadian rhythms are disrupted because our S.C.N.s receive light signals that are out of register with our previous protein accumulation cycles.

Additionally, circadian rhythm disruptions have been linked to obesity. Scientists compared light pollution data across the United States, and brightly-lit urban areas have significantly higher obesity rates. Further studies revealed that a hormone called leptin, responsible for appetite suppression, is regulated by the circadian clock. During the day when activity and energy requirements are increased, leptin expression decreases and thus we eat. When our circadian clocks are disrupted, leptin expression is lower, resulting in abnormally increased appetite and obesity.

A dive into the rhythmic inner-workings of our bodies can help explain why you have a bad night’s sleep after watching Netflix late at night, or why staying inside all day makes you less hungry. Dysregulated circadian rhythms may cause neurological disorders including Alzheimer’s disease, so further study of this symphony may offer potential treatments. Now, as the orchestra quiets and the conductor bows, scientists applaud the small compartment of our brain that keeps our bodies in sync.

Works Cited

Fonken, Laura K., and Randy J. Nelson. “The Effects of Light at Night on Circadian Clocks and Metabolism.” Endocrine Reviews, 1 Aug. 2014.

Froy, Owen. “Circadian Rhythms and Obesity in Mammals.” ISRN Obesity, 18 Nov. 2012.

“Jet Lag — Overview.” American Academy of Sleep Medicine.

Ko, Caroline H., and Joseph S. Takahashi. “Molecular Components of the Mammalian Circadian Clock.” Human Molecular Genetics, 15 Oct. 2006.

Nagourney, Eric. “Some Rhythmic Clues to Alzheimer’s.” The New York Times, 17 April 2001.

Sanders, Laura. “Out-of-Whack Body Clock Causes More Than Sleepiness.” Science News for Students, 3 Dec. 2019.

Fat Got Your Tongue?

我们通过发表论文来表彰学生 STEM 写作比赛的前八名获奖者。这是妮可·方(Nicole Fang)的作品。


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这篇文章由来自马里兰州北波托马克市理查德蒙哥马利高中的 16 岁的妮可·方(Nicole Fang)撰写,是学习网络首届 STEM 写作比赛的前八名获奖者之一,我们收到了 1,618 份参赛作品。

Fat Got Your Tongue?

Twenty-two million Americans stop breathing in their sleep without knowing. Snuggling up under their covers and swirling to the beautiful chaos of a new dream, their upper airway muscles stealthily relax, pinching off any aperture to oxygen. As breathing momentarily cuts off and reflexes violently kick in, their bodies unconsciously embark on a workout of raucous snoring, sporadic wake-ups and high blood pressure. For many struggling to get sufficient rest after a full night of what is known as obstructive sleep apnea, or O.S.A., their troubles may be attributed to one key culprit: fat tongues.

When you stick your tongue all the way out while looking in a mirror, can you see your entire uvula? If not, you may suffer from bearing an unusually large tongue, which falls back against the back of your throat as you sleep, effectively closing the airway and suspending respiratory activity for more than ten seconds.

While this increasingly common condition occasionally affects slim folks, overweight people make up around 70 percent of those with sleep apnea, according to a study published by Surendra Kumar Sharma in the journal Chest. With obesity continuing its unrelenting climb among adults today, weight loss is key for treating apnea.

Supporting this idea is a recent study published in the American Journal of Respiratory and Critical Care Medicine, where researchers gathered 67 people suffering from obesity and severe sleep apnea. Taking MRI scans of each participant’s pharynx as they lost around 10 percent of their body weight, the analysts observed tongue fat reduction to be the primary link between weight loss and apnea relief.

“No one really understands the relationship of obesity to sleep apnea, and no one knows much about tongue fat in general,” said Dr. Richard Schwab, senior author of the study. “But the correlation between the three is significant.” In his final statistical report, Dr. Schwab disclosed how losing weight reduced tongue fat by an average of 20 percent — a change greater than that of any other airway structure — and consequently, a 31 percent improvement in sleep apnea scores. Essentially, the slimmer the tongue, the more the disorder’s symptoms improved.

Acknowledging the agent and solution to this condition is critical in improving the quality of many lives, as apnea left untreated is a serious matter, according to Jonathan Jun, a sleep medicine specialist at Johns Hopkins. “We’re talking about car accidents in the daytime, lost productivity at work, mood swings, and falling asleep in class,” he said. In a more long-term perspective, sleep apnea also instigates heart disease, stroke and metabolic issues like diabetes.

The scariest part? You might not even know you have this prevalent disorder. With nine in 10 patients still undiagnosed, as reported by the American Academy of Sleep Medicine, Dr. Jun encourages everyone to simply avoid trouble by maintaining a healthy weight.

“It’s nothing new, really, to ask people to keep an eye on their weight,” he said. “But now, we know tongue fat is a risk factor for O.S.A., giving us a unique therapeutic target for future testing that we’ve never had before.”

Works Cited

Brody, Jane E. “Sleep Apnea Can Have Deadly Consequences.” The New York Times, 27 May 2019.

Lanese, Nicoletta. “A Fat Tongue May Be Blocking Your Airways While You Sleep.” LiveScience, 10 Jan. 2020.

“Losing Tongue Fat Improves Sleep Apnea.” ScienceDaily, 10 Jan. 2020.

Seppa, Nathan. “Wake-up Call for Sleep Apnea.” ScienceNews, 31 July 2008.

Wang et al. “Effect of Weight Loss on Upper Airway Anatomy and the Apnea Hypopnea Index: The Importance of Tongue Fat.” American Journal of Respiratory and Critical Care Medicine, 10 Jan. 2020.

A New Cancer Treatment That Is Faster, More Efficient and Less Risky Is Coming: Bacteria Bombs

我们通过发表论文来表彰学生 STEM 写作比赛的前八名获奖者。这是威廉·切斯尼(William Chesney)的作品。


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这篇文章由来自纽约布朗克斯伦理文化菲尔斯顿学校的15岁的威廉·切斯尼(William Chesney)撰写,是学习网络有史以来第一届STEM写作比赛的前八名获奖者之一,我们收到了1,618份参赛作品。

A New Cancer Treatment That Is Faster, More Efficient and Less Risky Is Coming: Bacteria Bombs

Bacteria are usually thought of as microscopic, disease-causing nuisances. They have long been seen as something that needs to be washed away with soap and warm water, or killed en masse with hand sanitizer. But what if these stigmatized organisms could be used to fight cancer, the stubborn killer devastating millions of people? Scientists at the University of California, led by Jeff Hasty, are working to genetically engineer bacteria that will infiltrate tumors and kill them. Combined with conventional treatment such as chemotherapy, researchers are hoping bacteria will be very effective at killing tumors and, further, stopping them from spreading. If proven safe and effective, this bacterial treatment could provide a vital victory in the war against cancer.

The immune system regularly patrols the body to find and destroy cancerous cells, but tumors can disguise themselves as normal cells by releasing a protein that tells the body to leave the tumor alone. And so, the tumor hides in plain sight, invading the healthy tissue around it. Tumors use blood vessels that branch all over the surface of the tumor to steal resources. Chemotherapy delivers cancer killing drugs to the bloodstream, but these drugs can only reach as far as the blood vessels reach, and most of the tumor’s blood vessels do not get to the center of the tumor.

This is where the bacteria, specifically salmonella, can come to the rescue. The scientists at the University of California have changed the DNA of bacteria to seek out these tumors, and populate them without invading the rest of the body. The bacteria settle in and start to replicate. But, like little bombs, they soon explode and release “a toxic cocktail” in the words of Dr. Sally Adee. The cocktail contains “three types of cancer-killing drugs: one that destroys cell walls, one that alerts the body’s immune system, and one that triggers cells to die” according to Dr. Adee. This allows the bacteria to attack the tumor directly in a localized way to keep the damage away from other healthy cells. Importantly, it also alerts the immune system to begin its offensive by destroying the tumor’s immunosuppressive proteins. In his Nature Reviews Cancer article, “Engineering the Perfect (Bacterial) Cancer Therapy,” the University of Massachusetts professor Neil Forbes said, “the immune system plays a complicated role in bacteriolytic therapy; it provides a mechanism to guide bacterial accumulation, but also impedes dispersion and efficacy.” Thus the body and these bacteria form a surprising team in their war against cancer.

Of course, this treatment has risks. The main danger is that the bacteria will become the enemy and infect the person. To prevent this, researchers have modified the bacteria to not only thrive in the environment of a tumor, but also to struggle to survive in healthy tissue. This is a revolution in the way people have thought about cancer treatment. Bacteria may soon join the list of unlikely allies in the fight against cancer, joining the ranks of debilitating chemicals and harmful radiation.

Works Cited

Adee, Sally. “Self-Destructing Bacteria Are Engineered to Kill Cancer Cells.” New Scientist, 20 July 2016.

Forbes, Neil S. “Engineering the Perfect (Bacterial) Cancer Therapy.” Nature Reviews Cancer, 14 Oct. 2010.

Williams, Thomas. “New Cancer Treatment? Scientists Have Programmed Bacteria to Kill Cancer Cells in Mice.” The Conversation, 21 July 2016.

Zimmer, Carl. “New Weapons Against Cancer: Millions of Bacteria Programmed to Kill.” The New York Times, 3 July 2019.

Sleep to Clean: A Prevention of Plaques That Lead to Alzheimer’s Disease

我们通过发表论文来表彰学生 STEM 写作比赛的前 11 名获奖者。这是乔斯琳·谭(Jocelyn Tan)的作品。


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本文由Jocelyn Tan撰写,15岁,来自新泽西州Bask Ridge的Ridge高中。,是学习网络第二届年度STEM写作比赛的前11名获奖者之一我们收到了3,741份参赛作品。

Sleep to Clean: A Prevention of Plaques That Lead to Alzheimer’s Disease

Our brain’s storage is like a teenager’s room — messy, cluttered and a fortress of personal memories. Scattered around are beloved belongings, such as your family heirloom or favorite cat, but imagine if suddenly these treasures disappeared, with only dust bunnies lying in their wake. Poof. Gone.

Despite seeming like deceptive magic, this is the blunt reality of an individual’s brain with Alzheimer’s, a daunting and currently incurable disease. With about 10 percent of people over the age of 65 diagnosed, it seems inevitable that it would affect individuals and families. But, what if this disease could be prevented through something simple — sufficient sleep?

Take a look inside the structure of a healthy brain. As they are created and destroyed, billions of neurons reside and correspond with each other through synapses. As new experiences feed into our brain every second, the synapses receive neurotransmitters that are responsible for the communication in our brain: seeing, thinking, remembering. However, in the brain of an Alzheimer’s patient, harmful proteins called amyloid-beta 42 block these synapses. Naturally produced by neurons, amyloid-beta proteins accumulate and lead to Alzheimer’s if not cleaned out fast enough by microglia cells, the cleaners for the brain. Over time, the rapid rate of amyloid-beta production causes the proteins to clump up into plaque. This unsettling change results in brain dysfunction; proteins like tau create neurofibrillary tangles that choke off the insides of neurons. Now, the once messy room is structurally and functionally destroyed by a hurricane. Looking to rapidly fix the chaos, microglia cells secrete inflammatory factors, resulting in prolonged inflammation and even the destruction of neurons.

In the past years, scientists began noticing a relationship between Alzheimer’s disease and sleep. Dr. Yo-El Ju evaluated patients in sleep apnea treatment. Following their successful treatment, she found that both the production and number of beta-amyloids had decreased. Laura Lewis, an assistant professor from Boston University who conducted a study on brain waves and sleep, said that the patients seemed “to have a change in their ability to clear proteins or waste products from their brain.” Hence, sleep has a vital role in the reduction of beta-amyloid plaques — the emerging signs of oncoming Alzheimer’s.

But, how come? In a separate study done by Dr. Maiken Nedergaard, it was discovered that the brain cleans waste two times faster when asleep. “So things like amyloid-beta, which are implicated in Alzheimer’s disease, seem to actually be removed more rapidly from the brain,” Dr. Lewis pointed out. Allowing microglia cells and other proteins to actively sweep out waste at much faster rates, sleep reshapes the untidy room in the brain, solidifying memories. With a healthy sleep routine, the fate of our brains could be deterred from Alzheimer’s disease.

There is still much to discover. As Dr. Lewis said, “I don’t know whether it’s that sleep increases clearance or whether sleep decreases the production of waste products.” Every step in the understanding of neurology can help uncover new preventions for Alzheimer’s, improving brain health for generations to come.

Works Cited

Alzheimer’s Association. “Alzheimer’s Facts and Figures.” Alzheimer’s Association.” Alzheimer’s Association.

Cunningham, Aimee. “Lack of Sleep Is Tied to Increases in Two Alzheimer’s Proteins.” Science News, 24 Jan. 2019.

Hamilton, Jon. “Deep Sleep Protects against Alzheimer’s, Growing Evidence Shows.” NPR, 17 Nov. 2020.

Konnikova, Maria. “Goodnight. Sleep Clean.” The New York Times, 11 Jan. 2014.

“What Happens to the Brain in Alzheimer’s Disease?” National Institute on Aging, 16 May 2017.